SKIN Panel investigates 6 main areas of Premature Skin Aging:
Collagen and elastin are the main proteins that give our skin its turgor and youthful complexion. Genes investigated in this section play a key role in production and maintenance of elasticity building blocks.
Genes: COL1A1, MMP1, MMP3, ELN
Antioxidant defence system is keeping our skin protected from harmful environmental free radicals and oxidative damage. Here we look at genes responsible for maintaining a firm protective shield.
Genes: SOD2, GPX1, NQO1, CAT
Photoaging is skin damage caused by ultraviolet radiation from the sun. It accelerates skin deterioration and affects all layers of the derma.
Genes: TYR, IRF4, GSTM1, GSTP1, MC1R, PIGU, XRCC1
Inflammation is our body’s natural defence mechanism that is triggered by aging. However, if chronic, it accelerates skin aging as cells experience increased stress levels. Genes investigated in this section are responsible for inflammatory response and activation.
Genes: IL – 1b, IL6, TNFa, IL13
Dry, cracking, easily irritated skin is one of the most common premature aging reasons. Here we target genes that are responsible for maintaining our skin sufficiently hydrated.
Genes: AQP3, FLG
Glycation is a spontaneous non – enzymatic reaction that is synonymous with skin aging. It is a known ’domino effect’ trigger of other skin aging processes. We investigate the gene programming this reaction.